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Village Dog Honduras

8 Village Dog Hondurass in the atlas. Every number on this page has a source.

8 Village Dog Hondurass in the Sniff Atlas. Population-genetic snapshot, Mendelian carrier frequencies from Donner 2023, and the data substrate's release version, sample sizes, and evidence tier on every claim.

What the atlas says about Village Dog Honduras

In the atlas, the village dog Honduras clusters consistently as village dog Honduras (100% of the 8 dogs here). Genetic diversity is high (mean heterozygosity 0.3685), reflecting either a mixed-breed cluster or breeds with broad genetic backgrounds. At the trait loci, FGF5 runs higher than the atlas average (100% here vs 64%); MC1R runs higher than the atlas average (94% here vs 62%).

Mean heterozygosity is 0.368, notably high, indicates broad genetic background. Only 8 dogs of this breed in the atlas, every individual contributes outsized weight to the breed's computed profile.

Closest genetic neighbors in the atlas: village dog Dominican Republic, village dog Peru Ica, village dog Peru Arequipa, village dog Peru Cusco, and village dog Brazil.

Genetic dimensions · CanVAS atlas

What the genome says about Village Dog Honduras

Computed from the 18,477 research dogs in the Atlas.

Dogs in the Atlas
8Founders
5 from Shannon, 3 from Hayward2016
Genetic diversity
0.37Diverse
Mean heterozygosity across the breed. Too few dogs in this breed (<20) to rank.
Cluster structure
Single tight cluster
Intra-breed RMS distance: 19.28
Nearest genetic relatives
  1. Village Dog Dominican Republic3.13
  2. Village Dog Peru Ica3.69
  3. Village Dog Peru Arequipa3.84
  4. Village Dog Peru Cusco4.30
  5. Village Dog Brazil4.43
Top-10 PC corrected Euclidean. Lower = closer.
Trait genetics
Allele frequencies at named morphology loci

Frequency of the alternate allele in this breed at each locus's representative SNP.

Body size
IGF175%
HMGA231%
SMAD269%
LCORL75%
STC281%
ADAMTS1756%
Leg length
FGF4·CFA1881%
FGF4·CFA1269%
Coat
RSPO263%
FGF5100%
KRT7175%
MC1R94%
Ear set
MSRB381%
Skull shape
BMP369%
SMOC263%
What you see when you look at a Village Dog Honduras

What does the genome say about how a Village Dog Honduras looks?

Village Dog Hondurass look the way they do because of a small set of fixed and near-fixed morphology genes that, taken together, define the visible breed. Each translation below pairs the gene with the trait an owner actually sees, the breed's allele frequency at that locus, and a one-clause causal phrase.

Size and build

IGF1 sits at 75% for the small-body allele. IGF1 is the gene that sets dog body size from Chihuahua to Great Dane. Intermediate frequencies typically keep a breed in the mid-sized range rather than tipping toward the larger working forms.

HMGA2 sits at 31%. HMGA2 is a chromosome-10 size locus that acts together with IGF1, and intermediate frequencies reflect partial commitment to the dominant size variant.

SMAD2 sits at 69% at the chromosome-7 height locus.

LCORL sits at 75% at the NCAPG/LCORL height locus on chromosome 3.

STC2 sits at 81%.

ADAMTS17 sits at 56%. ADAMTS17 is a body-size locus also linked to lens disorders.

Leg length

The FGF4 retrogene on chromosome 18 sits at 81%. This is the leg-length variant. The intermediate frequency means some dogs in this breed carry the short-legged allele and some do not.

The FGF4 retrogene on chromosome 12 sits at 69%, the chondrodystrophic variant.

Coat type, length, and color

RSPO2 sits at 63% for the furnishings variant. Furnishings (the eyebrow-and-mustache pattern seen in Schnauzers and Wheaten Terriers) vary across the population at this intermediate frequency, and visible expression depends on the specific allele combination each dog carries.

FGF5 is at 100% for the long-coat variant, which is why the breed's coat sits where it does on the long end of the dog coat-length spectrum.

KRT71 sits at 75% for the wavy/curly variant. Coat curl varies across individuals at this intermediate frequency, and visible expression is also influenced by modifier loci.

MC1R is at 94% at the representative SNP. MC1R controls the switch between red-to-gold and black-to-brown pigment, with the e/e homozygous genotype producing the gold-to-red spectrum by blocking eumelanin (black and brown pigment).

Ears

MSRB3 sits at 81% for the drop-ear allele, which is why ear set varies across the breed.

Skull shape

BMP3 sits at 69%, contributing to the breed's moderate, mesaticephalic head shape rather than the extreme brachycephalic form.

SMOC2 sits at 63%, contributing to the breed's moderate head shape.

The data behind this page

Where every number on this page came from.

This page draws on three primary data sources. Carrier frequencies for the Mendelian section come from Donner et al. 2023 (CC-BY-4.0). We grade these data at evidence Limited because the cohort is a direct-to-consumer ascertainment, which biases toward owners who chose to test their dogs. The panel also uses tag-SNP proxies for some variants rather than direct causal-variant assays. Limited is a study-design grade, not a quality grade: the Donner cohort is the largest open canine-genotype dataset in existence and we are grateful for it. We rate the confounding MEDIUM.

Population-genetic dimensions (heterozygosity, intra-breed PCA distance, nearest neighbors, trait-locus frequencies) come from CanVAS (Brundage 2026), harmonized through the Sniff Atlas. The exact release date and verification commit are pinned at the bottom of the page so a researcher can trace a number back to a specific snapshot. The disease-gene-variant graph comes from OMIA (Online Mendelian Inheritance in Animals; Nicholas, Tammen, and the Sydney Informatics Hub at the Sydney School of Veterinary Science, The University of Sydney; retrieved April 2026, DOI 10.25910/2AMR-PV70).

What this page does not yet have. Inheritance modes and per-disease penetrance evidence from Donner 2023 are now in the structured data for every variant the panel covers. Mondo, OMIM, Ensembl, and HGNC cross-references on gene pages remain pending — they arrive in December 2026 alongside the imputed 9.67M-variant CanVAS dataset via the OMIA SQL dump absorption. Until then, gene IDs carry NCBI Gene and OMIA phene URLs only; the wider human-homolog and disease-ontology cross-reference set fills in with that release.

How to cite this page. The computed dimensions on this page are derived from the open Sniff Atlas v1.0.1 (Gehring 2026, doi:10.5281/zenodo.20566358, CC-BY 4.0). Full citation formats including BibTeX, RIS, and CITATION.cff at sniff.world/cite.

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References
  1. Donner J, Anderson H, Davison S, et al. (2023). Frequency and distribution of 152 genetic disease variants in over 1,000,000 mixed-breed and purebred dogs. PLOS Genetics 19(2):e1010651. doi:10.1371/journal.pgen.1010651
  2. Brundage J, et al. (2026). CanVAS: a harmonized canine variant atlas. bioRxiv. doi:10.64898/2026.04.13.718238
  3. Nicholas, F.W., Tammen, I., & Sydney Informatics Hub. (2026). Online Mendelian Inheritance in Animals (OMIA) [dataset]. The University of Sydney. https://omia.org. doi:10.25910/2AMR-PV70 (retrieved April 2026).
Last updated
Sources: CanVAS (Brundage 2026) · Donner 2023 · OMIA