Nova Scotia Duck Tolling Retriever
52 Nova Scotia Duck Tolling Retrievers in the atlas. Every number on this page has a source.
52 Nova Scotia Duck Tolling Retrievers in the Sniff Atlas. Population-genetic snapshot, Mendelian carrier frequencies from Donner 2023, and the data substrate's release version, sample sizes, and evidence tier on every claim.
In the atlas, the Nova Scotia Duck Tolling Retriever clusters consistently as Nova Scotia Duck Tolling Retriever (100% of the 52 dogs here). At the trait loci, MC1R runs lower than average (16% here vs 62%); FGF4_retrogene_CFA12 runs lower than average (36% here vs 80%). Dogs here sit in a relatively sparse region of the atlas, fewer close neighbors than typical.
Closest genetic neighbors in the atlas: Australian Cattle Dog, Irish Water Spaniel, Briard, Old English Sheepdog, and Rat Terrier.
Median lifespan is 13.2 years, slightly longer than expected for the breed size (20.0 kg).
What the genome says about Nova Scotia Duck Tolling Retriever
Computed from the 18,477 research dogs in the Atlas.
- Australian Cattle Dog7.70
- Irish Water Spaniel8.89
- Briard9.09
- Old English Sheepdog9.34
- Rat Terrier9.39
Frequency of the alternate allele in this breed at each locus's representative SNP.
| IGF1 | 72% |
| HMGA2 | 34% |
| SMAD2 | 54% |
| LCORL | 100% |
| STC2 | 53% |
| ADAMTS17 | 63% |
| FGF4·CFA18 | 97% |
| FGF4·CFA12 | 37% |
| RSPO2 | 50% |
| FGF5 | 96% |
| KRT71 | 100% |
| MC1R | 16% |
| MSRB3 | 88% |
| BMP3 | 85% |
| SMOC2 | 95% |
What does the genome say about how a Nova Scotia Duck Tolling Retriever looks?
Nova Scotia Duck Tolling Retrievers look the way they do because of a small set of fixed and near-fixed morphology genes that, taken together, define the visible breed. Each translation below pairs the gene with the trait an owner actually sees, the breed's allele frequency at that locus, and a one-clause causal phrase.
Size and build
IGF1 sits at 72% for the small-body allele. IGF1 is the gene that sets dog body size from Chihuahua to Great Dane. Intermediate frequencies typically keep a breed in the mid-sized range rather than tipping toward the larger working forms.
HMGA2 sits at 34%. HMGA2 is a chromosome-10 size locus that acts together with IGF1, and intermediate frequencies reflect partial commitment to the dominant size variant.
SMAD2 sits at 54% at the chromosome-7 height locus.
LCORL is near-fixed at 100%, the NCAPG/LCORL height locus that is one of the strongest single contributors to canine body size.
STC2 sits at 53%.
ADAMTS17 sits at 63%. ADAMTS17 is a body-size locus also linked to lens disorders.
Leg length
The FGF4 retrogene on chromosome 18 is near-fixed in this breed at 97%. This is the leg-length variant. The breed is fully committed to the long-legged form rather than the short-legged Corgi-and-Dachshund body plan.
The FGF4 retrogene on chromosome 12 sits at 37%, the chondrodystrophic variant.
Coat type, length, and color
RSPO2 sits at 50% for the furnishings variant. Furnishings (the eyebrow-and-mustache pattern seen in Schnauzers and Wheaten Terriers) vary across the population at this intermediate frequency, and visible expression depends on the specific allele combination each dog carries.
FGF5 is at 96% for the long-coat variant, which is why the breed's coat sits where it does on the long end of the dog coat-length spectrum.
KRT71 is near-fixed at 100% for the wavy/curly variant. Coat curl phenotype varies across breeds at this fixation depending on modifier loci, and visible expression is not always curled even when the locus is fixed.
MC1R is at 16% at the representative SNP, leaving the breed in the black-to-brown coat range under the dominant E allele.
Ears
MSRB3 is at 88% for the drop-ear allele, the genetic basis of the breed's signature dropped ear set.
Skull shape
BMP3 is at 85%, contributing to the breed's brachycephalic skull shape.
SMOC2 is at 95%, the major locus contributing to the breed's brachycephalic face shape.
What genetic diseases do Nova Scotia Duck Tolling Retrievers carry?
From a panel of 250 Mendelian-disease variants screened in 1,054,293 dogs (Donner et al. 2023), Nova Scotia Duck Tolling Retrievers carry 7 of them at observable frequency. Carrier frequency is not clinical risk. Most recessive variants require two copies for disease expression; many dominant variants show incomplete penetrance. Read this as a population fingerprint of what's in the gene pool, not a per-dog prediction.
Where every number on this page came from.
This page draws on three primary data sources. Carrier frequencies for the Mendelian section come from Donner et al. 2023 (CC-BY-4.0). We grade these data at evidence Limited because the cohort is a direct-to-consumer ascertainment, which biases toward owners who chose to test their dogs. The panel also uses tag-SNP proxies for some variants rather than direct causal-variant assays. Limited is a study-design grade, not a quality grade: the Donner cohort is the largest open canine-genotype dataset in existence and we are grateful for it. We rate the confounding MEDIUM.
Population-genetic dimensions (heterozygosity, intra-breed PCA distance, nearest neighbors, trait-locus frequencies) come from CanVAS (Brundage 2026), harmonized through the Sniff Atlas. The exact release date and verification commit are pinned at the bottom of the page so a researcher can trace a number back to a specific snapshot. The disease-gene-variant graph comes from OMIA (Online Mendelian Inheritance in Animals; Nicholas, Tammen, and the Sydney Informatics Hub at the Sydney School of Veterinary Science, The University of Sydney; retrieved April 2026, DOI 10.25910/2AMR-PV70).
What this page does not yet have. Inheritance modes and per-disease penetrance evidence from Donner 2023 are now in the structured data for every variant the panel covers. Mondo, OMIM, Ensembl, and HGNC cross-references on gene pages remain pending — they arrive in December 2026 alongside the imputed 9.67M-variant CanVAS dataset via the OMIA SQL dump absorption. Until then, gene IDs carry NCBI Gene and OMIA phene URLs only; the wider human-homolog and disease-ontology cross-reference set fills in with that release.
How to cite this page. The computed dimensions on this page are derived from the open Sniff Atlas v1.0.1 (Gehring 2026, doi:10.5281/zenodo.20566358, CC-BY 4.0). Full citation formats including BibTeX, RIS, and CITATION.cff at sniff.world/cite.
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- Donner J, Anderson H, Davison S, et al. (2023). Frequency and distribution of 152 genetic disease variants in over 1,000,000 mixed-breed and purebred dogs. PLOS Genetics 19(2):e1010651. doi:10.1371/journal.pgen.1010651
- Brundage J, et al. (2026). CanVAS: a harmonized canine variant atlas. bioRxiv. doi:10.64898/2026.04.13.718238
- Nicholas, F.W., Tammen, I., & Sydney Informatics Hub. (2026). Online Mendelian Inheritance in Animals (OMIA) [dataset]. The University of Sydney. https://omia.org. doi:10.25910/2AMR-PV70 (retrieved April 2026).